Riluzol - Versionsgeschichte

190.91.117.36: Rfz7

2011-07-22T16:24:43Z

Rfz7

← Nächstältere Version		Version vom 22. Juli 2011, 16:24 Uhr
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	\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>		\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>
	<ref>Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]</ref><ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681]</ref> <ref>[http://chrisparsons.de/Chris/Acrobat/Rammes_2008_18493706.pdf The fraction of activated N-methyl-D-Aspartate receptors during		<ref>Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]</ref><ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681]</ref> <ref>[http://chrisparsons.de/Chris/Acrobat/Rammes_2008_18493706.pdf The fraction of activated N-methyl-D-Aspartate receptors during
	synaptic transmission remains constant in the presence of the		synaptic transmission remains constant in the presence of the glutamate release inhibitor riluzole J Neural Transm (2008) 115:1119–1126] </ref><ref>[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3000412/pdf/nihms-256277.pdf Glutamatergic Modulators: The Future of Treating Mood Disorders? Harv Rev Psychiatry. 2010 October ; 18(5): 293–303. doi:10.3109/10673229.2010.511059].</ref><ref> Anticonvulsant and sleep-waking influences of riluzole in a rat model of absence epilepsy European J Pharmcol. [http://www.sciencedirect.com/science/article/pii/001429999190503I 199 371 373 (1991)] </ref>
	glutamate release inhibitor riluzole J Neural Transm (2008) 115:1119–1126] </ref>
	<ref>[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3000412/pdf/nihms-256277.pdf Glutamatergic Modulators: The Future of Treating Mood Disorders? Harv Rev Psychiatry. 2010 October ; 18(5): 293–303. doi:10.3109/10673229.2010.511059].</ref>

Penarc: Rfz7

2011-07-22T15:17:04Z

Rfz7

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	synaptic transmission remains constant in the presence of the		synaptic transmission remains constant in the presence of the
	glutamate release inhibitor riluzole J Neural Transm (2008) 115:1119–1126] </ref>		glutamate release inhibitor riluzole J Neural Transm (2008) 115:1119–1126] </ref>
	<ref>[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3000412/pdf/nihms-256277.pdf Glutamatergic Modulators: The Future of Treating Mood Disorders? Harv Rev Psychiatry. 2010 October ; 18(5): 293–303. doi:10.3109/10673229.2010.511059. </ref>		<ref>[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3000412/pdf/nihms-256277.pdf Glutamatergic Modulators: The Future of Treating Mood Disorders? Harv Rev Psychiatry. 2010 October ; 18(5): 293–303. doi:10.3109/10673229.2010.511059].</ref>


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	\| bgcolor="#dfdfdf" \| '''Wichtige Übersichtsartikel'''		\| bgcolor="#dfdfdf" \| '''Wichtige Übersichtsartikel'''
	\| align="left" width="800"\|		\| align="left" width="800"\| <ref>Duprat et al [http://molpharm.aspetjournals.org/content/57/5/906.full.pdf+html The Neuroprotective Agent Riluzole Activates the Two P Domain K+ Channels TREK-1 and TRAAK ] Molecular Pharmacology 57:906-912 (2000)</ref>
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	\| bgcolor="#dfdfdf" \| '''Medline Suchterm/ Synonyme'''		\| bgcolor="#dfdfdf" \| '''Medline Suchterm/ Synonyme'''

Penarc: Rfz 4.u.5.

2011-07-22T15:02:25Z

Rfz 4.u.5.

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	\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''		\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''
	\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>		\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>
	<ref>Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]</ref><ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681]</ref>		<ref>Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]</ref><ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681]</ref> <ref>[http://chrisparsons.de/Chris/Acrobat/Rammes_2008_18493706.pdf The fraction of activated N-methyl-D-Aspartate receptors during
			synaptic transmission remains constant in the presence of the
			glutamate release inhibitor riluzole J Neural Transm (2008) 115:1119–1126] </ref>
			<ref>[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3000412/pdf/nihms-256277.pdf Glutamatergic Modulators: The Future of Treating Mood Disorders? Harv Rev Psychiatry. 2010 October ; 18(5): 293–303. doi:10.3109/10673229.2010.511059. </ref>


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	\| bgcolor="#dfdfdf" \| '''Struktur'''		\| bgcolor="#dfdfdf" \| '''Struktur'''
	\| align="left" width="800"\| [[Datei:Riluzole3D.gif\|thumb\|6-(Trifluormethoxy)-1,3-benzothiazol-2-amin		\| align="left" width="800"\| [[Datei:Riluzole3D.gif\|thumb\|490px\|6-(Trifluormethoxy)-1,3-benzothiazol-2-amin]]
	]]
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	\| bgcolor="#dfdfdf" \| '''Physiologie'''		\| bgcolor="#dfdfdf" \| '''Physiologie'''

Penarc: Rfz

2011-07-22T02:41:09Z

Rfz

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	\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''		\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''
	\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>		\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>
	Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission  I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]		<ref>Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]</ref><ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681]</ref>

	<ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons ~~</ref>~~ [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681] </ref>

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186.11.87.160: Rfz

2011-07-22T02:33:28Z

Rfz

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	\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''		\| bgcolor="#dfdfdf" \| '''Freie Übersichtsartikel'''
	\| align="left" width="800"		\| align="left" width="800"\| <ref>[http://www.if-pan.krakow.pl/pjp/pdf/2004/2_187.pdf Riluzole enhances the anti-seizure action of conventional antiepileptic drugs against pentetrazole-induced convulsions in mice] [http://www.nature.com/mp/journal/v7/n7/pdf/4001087a.pdf Antiepileptic drugs and agents that inhibit voltage-gated sodium channels prevent NMDA antagonist neurotoxicity Molecular Psychiatry 7:726- 733 (2002)]</ref>
			Mizoule J, Meldrum B, Mazadier M, Croucher M, Ollat C, Uzan A et al. 2-Amino-6-trifluoromethoxy benzothiazole, a possible antagonist of excitatory amino acid transmission  I anticonvulsant properties. Neuropharmacology 1995; 24: 767-773 [http://www.sciencedirect.com/science/article/pii/0028390885900115 Abstracts]

			<ref>Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons </ref> [http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018681 PLoS ONE 6(4): e18681. doi:10.1371/journal.pone.0018681] </ref>

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Penarc: 1te

2011-07-22T01:28:53Z

1te

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| align="left" width="800"| [[Datei:Riluzole3D.gif|thumb|6-(Trifluormethoxy)-1,3-benzothiazol-2-amin
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| bgcolor="#dfdfdf" | '''Physiologie'''
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== Bibliographie ==
<references />

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